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Barker, Ed
One or more keywords matched the following properties of Barker, Ed
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overview My Scopus ID is 7103411264. My NIH COMMONS name is BARKERE. Research Areas: Natural killer cells and HIV, innate lymphoid cells in HIV infection. My Faculty Profile at Rush University Medical Center: https://www.rushu.rush.edu/faculty/edward-barker-phd My Laboratory: https://www.rushu.rush.edu/research/departmental-research/microbial-pathogens-and-immunity-research/laboratory-edward-barker-phd My NCBI Bibliography: https://www.ncbi.nlm.nih.gov/myncbi/browse/collection/46831905/?sort=date&direction=descending My Scopus: https://www.scopus.com/authid/detail.uri?authorId=7103411264 Education: PhD, University of Illinois
One or more keywords matched the following items that are connected to Barker, Ed
Item TypeName
Concept Receptors, Natural Killer Cell
Concept NK Cell Lectin-Like Receptor Subfamily K
Concept NK Cell Lectin-Like Receptor Subfamily D
Concept Killer Cells, Natural
Academic Article Dysfunctional natural killer cells, in vivo, are governed by HIV viremia regardless of whether the infected individual is on antiretroviral therapy.
Academic Article Immunologic abnormalities associated with chronic fatigue syndrome.
Academic Article Interferon-alpha administration enhances CD8+ T cell activation in HIV infection.
Academic Article Killing of human immunodeficiency virus-infected primary T-cell blasts by autologous natural killer cells is dependent on the ability of the virus to alter the expression of major histocompatibility complex class I molecules.
Academic Article HLA-C and HLA-E reduce antibody-dependent natural killer cell-mediated cytotoxicity of HIV-infected primary T cell blasts.
Academic Article HIV modulates the expression of ligands important in triggering natural killer cell cytotoxic responses on infected primary T-cell blasts.
Academic Article Human T-cell leukemia virus type 1 (HTLV-1) p12I down-modulates ICAM-1 and -2 and reduces adherence of natural killer cells, thereby protecting HTLV-1-infected primary CD4+ T cells from autologous natural killer cell-mediated cytotoxicity despite the reduction of major histocompatibility complex class I molecules on infected cells.
Academic Article Role of natural killer cells in HIV pathogenesis.
Academic Article Lysis of endogenously infected CD4+ T cell blasts by rIL-2 activated autologous natural killer cells from HIV-infected viremic individuals.
Academic Article HIV-1 Vpr triggers natural killer cell-mediated lysis of infected cells through activation of the ATR-mediated DNA damage response.
Academic Article Degranulation of natural killer cells following interaction with HIV-1-infected cells is hindered by downmodulation of NTB-A by Vpu.
Academic Article Preparation and use of HIV-1 infected primary CD4+ T-cells as target cells in natural killer cell cytotoxic assays.
Academic Article The natural killer cell cytotoxic function is modulated by HIV-1 accessory proteins.
Academic Article The natural killer cell interferon-gamma response to bacteria is diminished in untreated HIV-1 infection and defects persist despite viral suppression.
Academic Article A Conserved HIV-1-Derived Peptide Presented by HLA-E Renders Infected T-cells Highly Susceptible to Attack by NKG2A/CD94-Bearing Natural Killer Cells.
Academic Article KIR3DL1 and HLA-B Density and Binding Calibrate NK Education and Response to HIV.
Academic Article Inability of natural killer cells to destroy autologous HIV-infected T lymphocytes.
Academic Article CD155 on HIV-Infected Cells Is Not Modulated by HIV-1 Vpu and Nef but Synergizes with NKG2D Ligands to Trigger NK Cell Lysis of Autologous Primary HIV-Infected Cells.
Academic Article HIV Latency-Reversing Agents Have Diverse Effects on Natural Killer Cell Function.
Academic Article Effect of a chimeric anti-ganglioside GD2 antibody on cell-mediated lysis of human neuroblastoma cells.
Academic Article Enteric bacteria induce IFN? and Granzyme B from human colonic Group 1 Innate Lymphoid Cells.
Academic Article Presence of Inflammatory Group I and III Innate Lymphoid Cells in the Colon of Simian Immunodeficiency Virus-Infected Rhesus Macaques.
Academic Article Presence of Natural Killer B Cells in Simian Immunodeficiency Virus-Infected Colon That Have Properties and Functions Similar to Those of Natural Killer Cells and B Cells but Are a Distinct Cell Population.
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  • Killer Cells Natural